New research from a team of genome scientists and DNA damage response experts breaks new ground in understanding the function of a protein currently limited in clinical trials for cancer treatments.
Stuart first determined that ATM alone enforces senescence both at 3 percent and 20 percent oxygen. Inhibiting ATM (or overexpressing TRF2) allowed cells to grow beyond their usual expiration date, ...
New research from a team of genome scientists and DNA damage response experts breaks new ground in understanding the function of a protein currently limited in clinical trials for cancer treatments.
Stuart first determined that ATM alone enforces senescence both at 3 percent and 20 percent oxygen. Inhibiting ATM (or overexpressing TRF2) allowed cells to grow beyond their usual expiration date, ...